Kinin Receptors & Endothelium-Dependent Responses

B2 kinin receptors: a major player in the release of nitric oxide and EDHF
P. M. Vanhoutte

Does EDHF contribute to the control of peripheral resistance?
H. Shimokawa, T. Matoba

The endothelium and the vascular protective effect of ACE inhibitors: what is the evidence from experimental and clinical studies?
L. Raij, M. Veronesi, M. S. Zhou

What is the relative importance of bradykinin-triggered release of nitric oxide vs EDHF in the therapeutic effects of ACE inhibitors in humans?
T. F. Lüscher, F. Cosentino

Surfing the Heart: European Board for Accreditation in Cardiology – ESC Newscast WebPath — The Internet Pathology Laboratory for Medical Education
C. Ceconi
Icons of Cardiology: Otto Frank and the “Frank-Starling” Relationship
A. M. Katz

H. Just

Bradykinin, a hypotensive and smooth muscle stimulating factor released from plasma globulin by snake venoms and by trypsin
M. Rocha e Silva and others

Role of endothelial cells in relaxation of isolated arteries by bradykinin
P. D. Cherry and others

Why are converting enzyme inhibitors vasodilators?
P. M. Vanhoutte and others

Kinins and endothelium-dependent relaxations to converting enzyme inhibitors in perfused canine arteries
J. V. Mombouli and P. M. Vanhoutte

Potentiation of endothelium-dependent relaxations to bradykinin by angiotensin I converting enzyme inhibitors in canine coronary artery involves both endothelium-derived relaxing and hyperpolarizing factors
J. V. Mombouli and others

Kinins mediate kallikrein-induced endothelium-dependent relaxations in isolated canine coronary arteries
J. V. Mombouli and P. M. Vanhoutte

Endothelium-dependent hyperpolarization caused by bradykinin in human coronary arteries
M. Nakashima and others

Role of endogenous bradykinin in human coronary vasomotor control
P. Groves and others

Abnormal flow-mediated epicardial vasomotion in human coronary arteries is improved by angiotensin-converting enzyme inhibition
A. Prasad and others

Contribution of bradykinin receptor dysfunction to abnormal coronary vasomotion in humans
A. Prasad and others